Viral inheritance in our DNA: new insights into Alzheimer's disease

New research shows that ancient viral residues in our DNA can trigger Alzheimer's disease.HIV drugs offer promising therapeutic approaches.

LONDON (BOLTWISE) - New research suggests that traces of ancient viruses in our DNA may play a role in the development of Alzheimer's disease.This discovery opens up new therapeutic approaches, particularly through the use of anti-HIV drugs, which are showing promising results in early trials.

Today's Deals on Amazon!˗ˏˋ$ˎˊ˗ The discovery that ancient viral residues in our DNA may play a role in the development of Alzheimer's has made the scientific community sit up and take notice.These human endogenous retroviruses (HERVs) are remnants of viruses that integrated into our genomes millions of years ago.Normally inactive, they can be reactivated by age or environmental factors, leading to a cascade of inflammatory processes in the brain.

Of particular note is the activity of some K-family HERVs in the brains of patients with Alzheimer's disease.These viral sequences produce proteins and RNAs that alert the immune system and cause chronic inflammation.This neuroinflammation is a central feature of Alzheimer's disease and can be accelerated by HERV reactivation.

A promising approach in the fight against this viral activity is the use of antiretroviral drugs that were originally developed for the treatment of HIV. In a pilot study, the active ingredient lamivudine was tested in patients with mild cognitive impairment.The results showed that lamivudine can reduce viral processes and reduce the toxic effect on nerve cells.

These discoveries could revolutionize the therapeutic approach to Alzheimer's disease.Although research is still in its infancy, the next steps are clear: large-scale clinical trials must be conducted to demonstrate the effectiveness of antiviral therapy.It is also important to decipher the exact mechanism of HERV damage to nerve cells.If this approach is successful, it will be the first time it will be possible to attack the cause of Alzheimer's disease directly, rather than simply alleviate symptoms.

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